ECG on presentation Click to enlarge |
- Bradycardia rate 30 bpm
- Nil atrial activity visible
- Normal axis
- Close to -30 deg with near isoelectric lead II
- Narrow QRS
- Similar morphology to old ECG below
- Late R wave transition
- T wave inversion leads V2-3
- New compared with old ECG
ECG from 1 year prior Click to enlarge |
Key features:
- Sinus rhythm, rate 60 bpm
- Left axis deviation
- PR prolongation
- QRS narrow
- Voltage criteria for LVH in lead aVL
- Junctional bradycardia
- New T wave changes
- Nil evidence atrial activity
- ACS
- New anterior T wave change but nil history of chest pain
- No significant ST segment changes
- Structural disease
- Cardiomyopathy
- Valvular disease
- Electrolyte abnormality
- Hypokalaemia - can cause ECG changes and clinical weakness
- Hyperkalaemia - can cause almost any ECG abnormality
- Drug toxicity
- Consider digoxin, calcium channel blockers, beta blockers
- Usually chronic toxicity in the elderly but can be acute
- May be seen with worsening of other co-morbidities especially renal failure / chronic kidney disease
- Environmental / Endocrine
- Hypothermia
- Thyroid Dysfunction - hypo or hyper
- Inflammatory / Auto-immune
- Pericarditis
- Myocarditis
- Sarcoidosis
- SLE
- Lyme disease
- Idiopathic / Age Related
- High vagal tone states
- Usually transient
- Vomiting
- Pain
- Under anaesthetic
References / Further Reading
Life in the Fast Lane
- Junctional Rhythm
- Chan TC, Brady WJ, Harrigan RA, Ornato JP, Rosen P. ECG in Emergency Medicine and Acute Care. Elsevier Mosby 2005.
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