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- Regular
- No P waves present
- Accelerated Idioventricular Rhythm (AIVR)
- Normal
- QRS - Prolonged (160ms)
- Discordant ST segment changes
- Excessive depression in lead V5 and excessive elevation V3 (just on -0.25 ST elevation / QRS depth)
Additional:
- LBBB Morphology
- Deep S in V1-3
- Broad R wave in lateral leads
- T waves massively disproportionate and peaked
- Note in leads V5-6 terminal portion of T wave becomes positive
The key abnormalities on this ECG are:
- AIVR
- LBBB with abnormal ST changes
- Massive peaked T waves
Broad differentials would include
- Ischaemia
- Drug toxicity
- Acid-base disturbance
- Electrolyte abnormality
At first glance I'd favour hyperkalaemia as the culprit and an urgent VBG was taken - K 8.8 mmol/L !
I unfortunately don't have a follow-up ECG and I expect following treatment sinus rhythm was restored and the T wave changes normalised. I would be interested to know if the LBBB was longstanding or secondary to the hyperkalaemia,
References / Further Reading
Life in the Fast Lane
Textbook
- Chan TC, Brady WJ, Harrigan RA, Ornato JP, Rosen P. ECG in Emergency Medicine and Acute Care. Elsevier Mosby 2005.
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