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- 96 bpm
- Regular
- Sinus rhythm
- Normal
- PR - Normal (~180ms)
- QRS - Normal (80ms)
- QT - 500-520ms (QTc Bazette 630-660 ms) Measured in lead II
- ST depression in leads II, III, aVF, V4-6
- Prominent U waves in leads V3-5
- Occur just before the p wave
- T-U fusion in all other leads
Interpretation:
- Marked QT Prolongation
- Features supportive of hypokalaemia / hypomagnesaemia
- U waves T-U fusion
- Variable QT measurement lead II vs lead V3 (end of T wave more easily identifiable)
- Potential contribution from sotalol - known QTc prolonging agent
What happened ?
Shortly after this ECG was performed the patient became unresponsive with the following ECG rhythm strip.
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Bloods revealed several metabolic alkalosis, hypokalaemia and hypomagnesaemia.
The patient was admitted to a critical care area for monitoring and correction of electrolyte / acid-base disturbance. In addition her sotalol was ceased due to its associated risk of QTc prolongation and she was commenced on metoprolol.
References / Further Reading
Life in the Fast Lane
Textbook
- Chan TC, Brady WJ, Harrigan RA, Ornato JP, Rosen P. ECG in Emergency Medicine and Acute Care. Elsevier Mosby 2005.
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