They are from a 74 yr old patient who I assume had multiple medical co-morbidities.
The patient presented having had several episodes of chest pain in the preceding 24 hours.
Check out our comments from the original case post here.
ECG 1
This first ECG was taken on arrival to the Emergency Department with the patient pain free.
ECG 1 - Pain free on presentation |
- 66
- Regular
- Sinus rhythm
- Borderline LAD (~ -30 deg)
- PR - Normal (~180ms)
- QRS - Normal (80ms)
- QT - 400ms (QTc Bazette ~ 420 ms)
- Minor ST depression lead III
Additional:
- T wave inversion leads II, III, aVF, V4, V5, V6
- Biphasic T wave leads aVR, V3
- Early precordial transition between V1 and V2
- Dominant R wave V2
- Prominent T wave lead V2
ECG 2
The patient then developed chest pain and the following ECG was recorded.
ECG 2- Taken during an episode of chest pain |
- 84
- Regular
- Sinus rhythm
- Normal (~20 deg)
- PR - Normal (~200ms)
- QRS - Normal (80ms)
- QT - 360ms (QTc Bazette ~ 415 ms)
- ST Elevation leads II (2mm), III (3mm), aVF (2mm), V4 (1.5mm), V5 (1mm), V6 (1mm)
- ST Depression aVR, aVL, V1, V2
- Note horizontal ST morphology in V2
Additional:
- Pseudo-normalisation of T waves leads II, III, aVF, V4, V5, V6
- Early precordial transition between V1 and V2
- Dominant R wave V2
- Prominent T wave lead V2
ECG 3
The episode of pain lasted only several minutes and resolved spontaneously.
The ECG below was taken 8 minutes after the second ECG with the patient now pain free.
ECG 3 - Patient pain free, taken 8 minutes following ECG 2 |
- 66
- Regular
- Sinus rhythm
- Borderline LAD (~ -30 deg)
- PR - Normal (~180ms)
- QRS - Normal (80ms)
- QT - 380ms (QTc Bazette ~ 410 ms)
- ST elevation leads III, aVF
- Reduced compered with ECG 2
Additional:
- T wave inversion leads II, III, aVF, V4, V5, V6
- Early precordial transition between V1 and V2
- Dominant R wave V2
- Prominent T wave lead V2
ECG series showing ischaemia with re-perfusion (ECG 1), subsequent re-occlusion (ECG 2) with infero-postero-lateral STEMI, and spontaneous re-perfusion (ECG 3).
What happened ?
The patient was immediately discussed with cardiology services. Treated with aspirin, clopidogrel, and placed on a heparin infusion and admitted to CCU. The patient remained pain free, troponin peaked at 12 hours, 4.8 (normal <0.05), and the patient was transfer the next day for angiography.
The angio showed:
- Right coronary: 98% stenosis --> stented
- Circumflex: 80% stenosis
- Left anterior descending: 80% proximal stenosis
- Left main: 20% proximal stenosis
- Left ventricle: Inferior hypokinesis with normal LV function
I'd recommend having a look at Dr Smith's ECG blog, links below, for some more great examples of re-perfusion / re-occlusion ECGs.
References / Further Reading
Dr Smith's ECG blog
- Pseudonormalisation of T waves
- Why are these precordial T waves large ?
- Spontaneous Re-perfusion Re-occlusion
- Prehospital series showing reperfusion
Life in the Fast Lane
Textbook
- Chan TC, Brady WJ, Harrigan RA, Ornato JP, Rosen P. ECG in Emergency Medicine and Acute Care. Elsevier Mosby 2005.
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